Arntl deficiency in myeloid cells reduces neutrophil recruitment and delays skeletal muscle repair

TLR2 signaling up-regulates GRK2 expression in neutrophils. ( A ) CXCR2

Towards improving the prognosis of stroke through targeting the circadian clock system

FOXO1 regulates TLR expression in neutrophils. (a,B) Bacteria were

Formylpeptide Receptors Mediate Rapid Neutrophil Mobilization to Accelerate Wound Healing

Molecular muscle physiology and pathophysiology – Lanner Lab

Myeloid cell diversification during regenerative inflammation: Lessons from skeletal muscle. - Abstract - Europe PMC

Representative images of the tibial anterior muscles, stained with

Microscopic evaluation for MyoD-positive nuclei in injured TA muscle

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ROS-activated CXCR2+ neutrophils recruited by CXCL1 delay denervated skeletal muscle atrophy and undergo P53-mediated apoptosis

DNA damage and the circadian clock

HSV-1 interactome

Macrophages recruited via CCR2 produce insulin-like growth factor-1 to repair acute skeletal muscle injury. - Abstract - Europe PMC

Immunohistochemistry of macrophages 2 (expressing mannose receptor) in